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Host-Pathogen Seminar Series 2007-2008
4pm First Floor BRT
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2007 |
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| September 17 |
Harry Mobley, Ph.D., Professor and Chair of Department of Microbiology and Immunology, University of Michigan.
Dr Mobley’s laboratory is interested in the molecular mechanisms of bacterial pathogenesis. The laboratory is studying virulence mechanisms of uropathogenic Escherichia coli and Proteus mirabilis that cause urinary tract infection, and Helicobacter pylori that causes gastritis and peptic ulcer disease.
"Genomic and Proteomic Basis for Vaccine Development against Urinary Tract Infection"
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| October 22 |
Aaron Mitchell, Ph.D., Harold S. Ginsberg Professor of Molecular Pathogenesis. Department of Microbiology, Columbia University.
Dr Mitchell’s interests are centered on the fungal environmental response pathways. His main focus is Candida albicans, the major invasive fungal pathogen of humans. C. albicans causes an array of infections in both immunocompetent and immunocompromised hosts, and their goal is to understand and combat virulence mechanisms. Dr Mitchell also works with the model yeast Saccharomyces cerevisiae, which offers unparalleled genetic and post-genomic resources. The goal of this work is to understand novel regulatory mechanisms.
"Control of cell surface features in the fungal pathogen Candida albicans"
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| November 5 |
David Woodland, Ph. D., Member, Trudeau Institute
NOTE: This seminar will start at 3:30pm.
The goal of Dr Woodland’s research program is to understand T cell memory in the lung with a view to vaccine development. Studies focus on several respiratory pathogen models in mice, including influenza virus, Sendai virus, a murine ?-herpesvirus (MHV-68), and M. tuberculosis.
"T cell memory to respiratory virus infections"
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| 2008 |
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| January 14 |
David Engman. Ph. D, M.D., Associate Professor of Pathology and Microbiology-Immunology, NorthWestern University.
Dr Engman’s research is focused on 3 areas: (i) organelle biogenesis in trypanosomes, (ii) pathogenesis and treatment of inflammatory heart disease and (iii) diagnosis of human genetic and infectious diseases.
"Trypanosomiasis: Role of the Flagellum in Biology and Disease"
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June 2
4:30pm |
Vojo Deretic. Ph. D., Department Chairperson. Molecular Genetics and Microbiology, University of New Mexico HSC.
Autophagy is as a fundamental biological process defined as a cytoplasmic homeostasis pathway whereby cytoplasm portions get sequestered by membrane for delivery to lysosomes. This leads to removal of damaged or surplus organelles and turnover of stable, long-lived macromolecules. Autophagy has been previously implicated in both health-promoting and disease-associated states in cancer, neurodegeneration, development, and aging. Autophagic degradation is a major effector of innate and possibly adaptive immunity mechanism for direct elimination of intracellular microbes and other aspects of immunity.
"Autophagy: from cytoplasmic homeostasis to immunity"
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